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The connection to FOXO3 is direct and functionally significant: both FOXO3 and NRF2 independently activate GCLC, GCLM, and GPX1 expression through separate transcriptional pathways FOXO3 through forkhead response elements (FHREs) and NRF2 through AREs

[DOI] [PubMed] [Google Scholar] 83.Schiattarella G.G., Hill J.A

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Differently, a marked depletion of GSH and ATP levels, accompanied by a profound increase in TBARS and ROS production, are associated with hepatocyte necrosis (Figure 4) [9]