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Additionally, mitochondrial-derived peptide-c (MOTS-c) has been shown to reverse mitochondrial dysfunction and reduce oxidative stress, thereby alleviating metabolic reprogramming, decreasing beta-oxidation, and increasing glycolytic flux influenced by abnormal metabolism during myocardial infarction [464]

doi: 10.1007/978-3-031-89525-8_6

Those groups pretreated with scopolamine followed by Nle1-tetrapeptide, or Nle1-tripeptide, required longer durations to locate the platform, however their search patterns were effective

Diabetes Mellitus, Race, and Effects of Omega-3 Fatty Acids on Incidence of Heart Failure Hospitalization