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Cells 12, 1671

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While samples for this assay were chosen to have undetectable levels of AMG 966, an anti-AMG 966 monoclonal antibody was also used in parallel as a detection reagent to ensure binding signal in the assay was not due to AMG 966
Upon exploration, we observed that the stomach and omentum were severely adhered to the diaphragmatic defect

doi:10.1056/NEJMcibr1315544

We propose that these terminals may possess independently regulated glycolytic enzymes and mitochondrial networks, allowing them to generate ATP rapidly in response to high-frequency firing without solely depending on axonal transport from the cell body
