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These changes produce spatial gradient differences in hepatic lobules, and affect the changes in gene expression profiles of hepatocytes, endothelial cells, etc

Myristoylated FSP1 was recruited to the plasma membrane, and catalyzed the reduction of ubiquinone (CoQ 10 ) by using NAD(P)H to form ubiquinol that functioned as a radical-trapping antioxidant to terminate LPO and finally suppressed ferroptosis

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Therefore, further elucidating the effect of cellular ferroptosis on delayed wound healing of diabetic ulcers, and exploring the specific molecular mechanism of ferroptosis-related signaling pathways in the occurrence and development of diabetic ulcers are of great significance for noval clinical therapeutic ideas for diabetic ulcers