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While direct evidence linking the RARA::RXRA and RARA::RXRG complexes to CCL11 expression in the CNS is currently not well established, it is plausible to speculate that retinoic acid signaling through these complexes could inhibit CCL11 expression in specific brain conditions

There is now extensive research demonstrating that oxidative stress can be a product of neurotransmitter dysfunction (e.g., hyperactivity of dopamine) and dysfunctions of glial cells, the endocrine system, and mitochondria

Thus, it is difficult to determine a causal relationship between the levels of GSH and clinical outcomes

Glutathione peroxidase 4 (GPX4), an antioxidant protein that is highly expressed in the brain, has been suggested as a likely target of DAQ modification (8)

This can be stated given the particular counteracting effect on convulsions (i.e., insulin hypoglycemic convulsions [62], and paracetamol over-dose-convulsions, progressive hepatic encephalopathy with generalized convulsions [61]), the improved conscious/unconscious/death ratio in trauma brain injury mice [78], attenuated/counteracted cerebellar ataxia, fore and hind limbs disabilities in cuprizone rats [80], and counteracted sedation/unconsciousness (diclofenac [108], ibuprofen [107])

Mechanism of ferroptosis resistance in cancer cells