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This process activates downstream genes (e.g., the caspase cascade) and promotes complex II formation, inducing apoptosis (199)

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However, the same dose in humans yielded plasma concentrations that were almost entirely below the detection limit at nearly all times, thereby precluding the calculation of pharmacokinetic parameters

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A plethora of research work has been carried out on this nucleus-bound DCM biomarker and also its multiple molecular pathways which are interconnected and have been characterised (PI3K/AKT/MAPK) although its inhibition had not produced an effective anti-DCM outcome that could be utilized as a valid therapeutic target

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