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found that emodin can inhibit the growth of HepG2 cells in a dose - and time-dependent manner induce cell S phase and G2/M phase stagnation promote apoptosis of HepG2 cells and thus control the migration and invasion of HepG2 cells

They exhibit better encapsulation efficiencies, reaching up to 90% for lipophilic pharmaceutical compounds (PCs)

Drug-induced liver injury: premarketing clinical evaluation, 2009

Oral glucose tolerance tests performed in clinical trials showed no significant changes in glucose handling, and fasting insulin levels remained stable throughout treatment periods

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Low content of the natural ocular anti-angiogenic agent pigment epithelium-derived factor (PEDF) in aqueous humor predicts progression of diabetic retinopathy