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The demonstration of increased incidence of culturable bacteria in the mesenteric lymph nodes of patients with intestinal obstruction supports the concept of BT in patients with severe physiologic stress and mucosal injury and would suggest that BT does not take place without mucosal damage and/or inflammation or trauma

This review summarizes the generally accepted mechanisms of APAP hepatotoxicity and highlights the key signaling pathways as targets for AILI therapeutic interventions

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