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Studies have shown that ferroptosis is linked to elevated prostaglandin-endoperoxide synthase 2 (PTGS2) expression and prostaglandin E2 (PGE2) secretion, which accelerates arachidonic acid metabolism, releasing inflammatory mediators and activating the inflammatory response [11]

These results suggested that TC- P

Neuron-like activation of microglia and astrocytes induces pro-inflammatory mediator expression including such as cytokines (interleukin (IL)-1, tumor necrosis factor (TNF)-, complementary components, acute-phase protein (Smith et al., 2012), which can stimulate inducible nitric oxide synthase (iNOS) and NO production with abnormal phagocytic activity (Lyman et al., 2014) (Figure 2)

Furthermore, GHD may occur as isolated GHD or with multiple pituitary hormone deficiencies

O., Haskell, C

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