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This is marked by increased PD-L1 expression, impaired T cell activation, and resistance to immune-mediated cell death, underscoring PTEN deficiency as a key factor in GBM immune escape

In response to inflammatory stimuli, immune cells reconfigure metabolism through cellular responses mediated by NF-B signaling

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Moreover, an in vitro hemolysis assay showed that GOx/CuSAE had minimal influence on the erythrocyte membrane, as indicated by negligible absorbance of released hemoglobin in the supernatant (Supplementary Fig

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