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Although mounting evidence suggests that complex neurochemical, behavioral, and pharmacological interactions exists between DACB1 receptor systems in the brain, few studies have directly investigated the interplay between these systems, and next to nothing is known about how a history of heavy chronic exposure to 9 -THC impacts subsequent stimulant neurobiology in both sexes

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it just means it hasn't been formally proven

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Three biosynthetic pathways regulate the cellular NAD + level: (1) the de novo synthesis pathway, (2) the PreissHandler pathway, and (3) the salvage pathway [15]