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Description
This hypothesis is a result of the discovery that GPX4 inactivation-induced ferroptosis is caused mostly by nonmitochondrial lipid peroxidation [66]
ERGO needs GSH for biosynthesis
Intracellular concentrations range from 12 mM in most cells to ~10 mM in hepatocytes, which also export GSH into plasma and bile
Non-enzymatic antioxidants, on the other hand, act as scavengers that neutralize free radicals before they can cause cellular damage
Human apolipoprotein A-I gene expression increases high density lipoprotein and suppresses atherosclerosis in the apolipoprotein E-deficient mouse
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