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doi: 10.1097/00004836-199704000-00013

Ex vivo mouse retinas exposed to 4-hydroxynonenal (4-HNE) induce RPE cell death through increased lipid peroxidation [180]

Consequently, there is an increase in intestinal permeability and translocation of intestinal flora, leading to the release of enterogenic endotoxins into the bloodstream which act on the original organ via the circulatory system, further exacerbating deterioration of the intestinal organ
Loss of intestinal alkaline phosphatase leads to distinct chronic changes in bone phenotype
S5g) Quantification of the antibody levels (Fig

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