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Preclinical research commonly evaluates Semax for its potential influence on pathways associated with neuronal plasticity and cellular resilience, including modulation of neurotrophin-related markers and neurotransmitter-linked signaling in controlled experimental settings

Microglial glutamate uptake is coupled to glutathione synthesis and glutamate release

FMT may correct dysbiosis, which plays a critical role in the pathogenesis of SAH [87]

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More frequent sampling, particularly in the early stages of the hospitalization, may generate more meaningful data that can compare the temporal profiles of candidate biomarkers to one another