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10.1177/1535370214558024 55 SaltiT.BraunsteinI.HaimovichY.ZivT.BenharM

P53 activation can sensitize cells to ferroptosis after ROS-induced stress by repressing the expression of GPX4 and system Xc component SLC7A11, which limits GPX4 substrate availability [61, 63,64,65,66]

In these two repair mechanisms, a variety of enzymes are involved, including DNA polymerase, DNA exonuclease, DNA ligase, etc., which cooperate to complete the repair of DNA

Acute and sub-chronic (28-day) oral toxicity profiles of newly synthesized prebiotic butyl-fructooligosaccharide in ICR mouse and Wistar rat models

[PubMed] [Google Scholar] 91.Behairy A., El-Sharkawy N.I., Saber T.M., Soliman M.M., Metwally M.M.M., Abd El-Rahman G.I., Abd-Elhakim Y.M., El Deib M.M