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As a competitive inhibitor of xCT, it promotes GSH consumption while inhibiting the expression of the antioxidant H2S-producing enzyme cystathionine-synthase (CBS) to achieve oxidative stress toxicity

Your medical team should review your goals, medical history, medications, labs, and risk factors before discussing any therapy

Huckelhoven and Kogel, 2003)

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Mutations in mitochondrial DNA caused by ROS can exacerbate mitochondrial dysfunction, creating a vicious cycle of oxidative stress and impaired sperm function

Blood cultures and additional sepsis evaluation are performed as clinically warranted