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Direct injection of EP4 agonist CP-734432 to ovariectomized rats was also sufficient to decrease BMAd number and size while promoting de novo bone formation at both rBMAd- and cBMAd-containing sites (6)

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The function of the fatty-acid oxidation pathway in fibroblast cultures and biomarkers for abnormalities in non-mitochondrial fatty-acid metabolism were not consistently abnormal across the subgroup of ASD children, consistent with the notion that the abnormalities in fatty-acid metabolism found in this subgroup of children with ASD were secondary to TCAC abnormalities