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Thus, the modulatory role of Gepon appeared to be targeted toward reducing excessive pro-inflammatory activity rather than enhancing anti-inflammatory outputs in myeloid cells [1]

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10.1183/13993003.02022-2022 3 AdlerK

Furthermore, the same receptor expressed in two distinct cell types can be endocytosed through different mechanisms due to differences in GRK and arrestin expression: either through clathrin-coated vesicles or caveolae

Ying Liu: Formal analysis, Writing review & editing
