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Among these, selenium-dependent glutathione peroxidase 4 (GPX4) is crucial for preventing ferroptosis by detoxifying phospholipid hydroperoxide into the corresponding phospholipid alcohol at the expense of glutathione (GSH) [12,13,14,15]

We therefore focused on the swapped region encompassing residues 1191 to 1270 forming the TM helices 16 and 17

However, the relative contribution of NRF2 target genes to NRF2-dependent cancer cell proliferation is currently unknown

During ischemia, succinate accumulates significantly within the mitochondrial matrix

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