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10.1016/j.intimp.2020.106261 111 LiF.WiegmanC.SeiffertJ

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[70] found in vivo and in vitro studies that GLP-1 and its analog exendin-4 were able to induce an increase in ABCA1 mRNA, increase the expression of apo AI mRNA and promoter, thereby increasing apo AI secretion, indicating that GLP-1 in hepatocytes may mediate the outflow of intracellular cholesterol to apo AI through the ABCA1 pathway

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Under certain conditions, competition between nitration and phosphorylation on a single tyrosine residue may completely disrupt a complex chain of signal transduction, as recently shown in primary rat hepatocytes

Grnvold, B