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doi: 10.1210/clinem/dgz263 9 QiXYunCPangYQiaoJ

GPX4 mutations cause SSMD-like symptoms Smith et al

163,164 It operates in BER and autonomously controls the oxidationreduction status of transcription factors

In Silico docking, molecular dynamics and binding energy insights into the bolinaquinone-clathrin terminal domain binding site

Aging Cell 20 , e13316 (2021)

The exacerbated impairments in relaxation and pressure development in G6PDX mice were rescued by antioxidant treatment, but not by ribose supplementation, demonstrating that the deficit was due to a deficiency in antioxidant defenses rather than a ribose deficiency resultant from the nonoxidative phase of the pentose phosphate pathway