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When patients with JakobCreutzfeldt disease underwent PET imaging with [ 11 C] d-l-deprenyl and [ 18 F]fluorodeoxyglucose ([ 18 F]FDG), increased astrogliosis and decreased cerebral glucose metabolism were observed in the frontal, occipital and parietal cortices and cerebellum 351,352
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In order to identify drivers of lapatinib tolerance, we focused on clusters 2 and 9 which comprised transcripts exhibiting sustained increased expression throughout the time course of the experiment
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