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Description
Tetrasulfide bond boosts the anti-tumor efficacy of dimeric prodrug nanoassemblies
The ozone-dependent ROS-mediated fatty acid oxidation leads to the formation of lipid ozonization products (LOPs), which act as signal transducers by triggering ROS signaling and therefore mitohormetic processes
doi: 10.3390/nu14173474 4
ROS Excess and Antioxidant Mechanisms As already mentioned, oxygen is essential for life, but its intermediaries can be a source of disease, through an uncontrolled production of ROS and more specifically of free radicals (FR) that damage the structure of organelles and macromolecules (lipids, proteins, carbohydrates and nucleic acids) [12,13]
Structurally, the mechanical component is formed by intestinal epithelial cells and vascular endothelial cells joined by tight junctions, adherens junctions, and cadherin proteins, thereby ensuring selective nutrient transport
the metalloproteases via Matrix metallopeptidase 9 (MMP-9) and MMP-2
