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In the rat retina, IGF-1 can induce retinal ganglion cell (RGC) survival and axon growth by activating phosphatidylinositol 3-kinase (PI3K) and mitogen-activated protein kinase (MAPK)/extracellular-signal regulated kinase (ERK) pathways (Dupraz et al., 2013

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This suggests that the mutation causes a significant conformation change

Once in the cytoplasm, intracellular thioesterases rapidly convert SAG to GSH

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However, if their degradation or biliary excretion are impaired, LPS can reach the systemic circulation, where they bind to Toll-like receptor 4 (TLR4) on leukocytes, endothelial cells, and platelets, causing arterial inflammation