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Together, these findings demonstrate that, in contrast with acute exposure, chronic disruption of iron homeostasis with consequent GSH depletion remodels cellular redox homeostasis over time, inducing a state we term chronoferroptosis: a persistent ferroptotic adaptation characterized by coordinated alterations in iron-handling and antioxidant defense proteins that may represent early vulnerability to neurodegenerative pathology

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Hydroxocobalamin acts as a crucial cofactor in various metabolic processes, supporting the conversion of homocysteine to methionine, which is essential for DNA synthesis and cellular function

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How a dog's own antioxidants like glutathione, SOD, and CoQ10 protect cells, why these defenses weaken with age, and when extra support may be worthwhile.