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Methylmalonic aciduria and homocystinuria type C protein convert cyanocobalamin into the active methylcobalamin and adenosylcobalamin forms in cells, as long as there is no remethylation disorder cblC in methylmalonic aciduria and homocystinuria type C protein, cblF and cblJ in the protein integral membrane of lysosomal cobalamin transport-escort protein LMBD1 and ATP-binding cassette subfamily D member 4required for lysosomal release of transport of cobalamin

A: PROBALAMIN is generally well tolerated

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Tissue-Repair Research: Early chondrocyte studies suggested proteoglycan synthesis modulation, though evidence is confined to animal and in-vitro work

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