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When TNF- binds to its receptors TNFR1/2, it activates a TRAF2-mediated JNK-dependent kinase cascade, sequentially activating MKKs, MAPKs, and ultimately p38 MAPK

Obvious vacuoles, very small amounts of sperm, a small number of early spermatogenic cells, extremely disordered cell arrangement, reduced cell layer number, the disappearance of cell polarity and a reduced Johnsen score were observed

This ATP deficiency disturbs multiple ATP-dependent processes, such as electrolyte transport, substrate utilization, and contractile protein interactions, resulting in impaired myocardial contractility

[DOI] [PMC free article] [PubMed] [Google Scholar] 67.Sahebkar A., Mohammadi A., Atabati A., Rahiman S., Tavallaie S., Iranshahi M., Akhlaghi S., Ferns G.A., Ghayour-Mobarhan M