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In the sections that follow, we build on this conceptual framework to examine how major proteostasis machineries contribute to cellular and organ-level resilience and how their failure drives pathology across cardiovascular, cancer, metabolic, neurodegenerative and autoimmune disorders and highlight therapeutic strategies aimed at reinforcing proteostasis resilience

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adopted a cascade reaction between the degradation of uric acid and timely elimination of H 2 O 2 using uricase and CAT-like nanozymes-Pt/CeO 2 nanozymes (Fig