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The fourth group was cultured in hypoxia, incubated with PBS and the probe DSNBD-GSH in sequence before being imaged (Figures 5jl)

Its primary mechanism is the upregulation of telomerase, the enzyme responsible for maintaining and extending telomeres, the protective end-caps on DNA strands

doi: 10.1016/j.schres.2016.07.010

High nitric oxide production in human paranasal sinuses

Cellular adaptation mediated through Nrf2-induced glutamate cysteine ligase up-regulation against oxidative stress caused by iron overload in beta-thalassemia/HbE patients

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