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Considering c-MET in aspects of cell signaling network through cross-talking with EGFR, VEGFR-2 and other signaling molecules, it is necessary to develop antibodies that are able to inhibit simultaneously two signaling cascades to achieve maximal therapeutic activity [77, 119]

Providing precursor molecules such as N-acetyl-cysteine or glutathione in its reduced form or as S-acetyl-glutathione in which the acetyl group protects the compound from being degraded in the GI tract are ways to boost levels of this crucial compound.

Collier et al., 2017

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