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[DOI] [PMC free article] [PubMed] [Google Scholar] 168.Wood Z.A., Schroder E., Robin Harris J., Poole L.B

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Mechanism of Action AOD9604 has been investigated for its interaction with beta-3 adrenergic receptor pathways in adipose tissue

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While increased UCB levels are so far the only identified outcome in Gilberts Syndrome, we cannot exclude that UGT1A1 mutations might exhibit other effects on human health

The observations from our animal studies prompted us to examine whether dysregulated cellular glutathione homeostasis could render hepatocytes susceptible to TNF-induced cell death