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[DOI] [PubMed] [Google Scholar] 21.Snezhkina A.V., Kudryavtseva A.V., Kardymon O.L., Savvateeva M.V., Melnikova N.V., Krasnov G.S., Dmitriev A.A

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Moreover, during zinc deficiency, enzymes such as glycosyltransferases and proteases are impaired, resulting in faulty receptor trafficking, extracellular matrix disintegration and hypoglycosylated proteins, defects that underlie congenital disorders of glycosylation 137

This strategy not only maximizes the therapeutic potential of each cytokine but also offers a versatile approach to incorporating gene delivery and recombinant cytokines as tools to enhance anti-cancer treatment efficacy