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Notch pathway up-regulation via curcumin mitigates bisphenol-A (BPA) induced alterations in hippocampal oligodendrogenesis

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Conversely, BACH1 knockdown not only inhibited BPC157-induced HUVEC proliferation and tube formation but also blocked the expression of these proangiogenic factors, confirming that BACH1 is the pivotal downstream effector of the BPC157-FBXO22 axis

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[1] [2] The compound has been found to potently improve cognitive function in animal models of Alzheimer's disease-like mental impairment

Endometriotic tissue is unable to respond to progesterone exposure, a phenomenon known as progesterone resistance