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However, the specific knockout of KLF4 in AdvSca1-SM cells induces it to differentiate into a more protective cell phenotype, and enhances plaque stability by changing plaque composition (such as reducing necrotic core and increasing fiber cap thickness), thus inhibiting atherosclerosis from developing toward instability (106)

Sayin SI, Wahlstrom A, Felin J, Jantti S, Marschall HU, Bamberg K, Angelin B, Hyotylainen T, Oresic M, Backhed F

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Furthermore, the maximum RMSF fluctuations in the apo form have occurred at the residues of Ser96, Arg209 and Lys292 (Fig