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An interesting study addressing this issue showed that patients with major depression that included psychosis had more differentially expressed proteins associated with energy metabolism, whereas patients with depression without psychosis had changes in proteins associated with cell growth and maintenance, although 53.7% of the altered proteins overlapped ( That alterations in mitochondria bioenergetics pathways contributed to the pathophysiology of depression also raise the possibility of developing mitochondrial biomarkers that can illustrate a better therapeutic approach to the treatment of depression

It also exerts potent anti-inflammatory effects by downregulating cytokines like TNF- and IL-6, while providing cytoprotection (shielding cells from toxins) and neuroprotection (guarding neurons from damage, potentially aiding conditions like Parkinson's in animal models)

Tumor imaging and therapy using radiolabeled somatostatin analogues