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When the oxidative stress state persists and the damage progresses, senescent cells enter a senescent state marked by p16 upregulation, which consequently inhibits the kinases CDK4 and CDK6, thereby maintaining the stagnant state of the cell cycle [133, 134]

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Together, these peptides create a comprehensive anti-aging approach: GHK-CU addresses visible signs of aging through tissue regeneration and skin health, while Epithalon works at the cellular level to support healthspan and biological age optimization