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Together, these pro-oxidant actions affect the cellular thiolstat and, especially under the conditions of sufficient OS, may also cause apoptosis

Moskalev A, Aliper A, Smit-McBride Z, Buzdin A, Zhavoronkov A (2014) Genetics and epigenetics of aging and longevity

however, genetics, cytokines, growth factors, hormones, such as estrogens and progesterone and/or their respective receptors, environmental and epigenetics, and the excessive synthesis of extracellular matrix (ECM), have been associated with the pathogenesis of UF (Flake et al., 2003

Vanadium derivative exposure promotes functional alterations of VSMCs and consequent atherosclerosis via ROS/p38/NF-kappaB-mediated IL-6 production

Exogenous selenium promotes cadmium reduction and selenium enrichment in rice: Evidence, mechanisms, and perspectives