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Yang, D., Hu, C., Wang, X., Shi, G., Li, Y., Fei, Y., Song, Y., & Zhao, X

Deletion of LCN2 in late osteoblasts and osteocytes reduces their intracellular iron accumulation, enhances antioxidant defenses, and mitigates ferroptosis susceptibility in vivo To investigate the role of LCN2 in regulating biological processes within late osteoblasts and osteocytes vivo, we generated Dmp1-Cre

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Finally, our findings are based on a manufacturing approach that depletes leukapheresis products of CD14+ monocytes and CD25+ Tregs, followed by positive selection of CD62L + T cells, and should not be generalized to other manufacturing approaches without further experimental support