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Yet, it became clear quite quickly that severe cases of COVID-19 comprise systemic endothelial dysfunction and inflammation in respiratory and non-respiratory organs that result from deregulated type I interferon (IFN I) signalling ( Several studies identified DNA-sensing pattern recognition receptors (PRRs) as drivers of IFN I-driven inflammation and massive cell death in severe COVID-19: cyclic GMPAMP synthase (cGAS), absent in melanoma (AIM)2, nucleotide-binding oligomerization domain (NOD)-like leucine-rich repeat (LRR) and PYRIN domain containing (NLRP)3, receptor for advanced glycation end products (RAGE), and Toll-like receptor (TLR)9 ( Figure 1 , see refs ( Figure 1 8 ] activating IRF3

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BPC-157's high proline content and TB-500's absence of disulfide bonds give the combined formulation robust stability under standard reconstitution and storage