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This paradoxical vulnerability indicates that GSH-dependent redox homeostasis, while essential for normal cellular function, can become a critical liability in cancer cells with dysregulated GSH metabolism

Funding This research did not receive any specific grant from funding agencies in the public, commercial or not for profit sectors

These data, in corroboration with the rate of release, safety, and stability of DNZ-NLP in vitro (Figs

2024;3:e70000

Phng php ln men mang li nhiu u th: glutathione thu c c cu trc phn t ng nht, khng cha tp cht t qu trnh tng hp ha hc, v n nh hn trong sut thi hn s dng

C th, Glutathione c kh nng c ch qu trnh hnh thnh sc t Melanin, ngn nga nguy c hnh thnh cc m sc t trn da nh thm mn, nm, tn nhang