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Iron-dependent mitochondrial dysfunction contributes to the pathogenesis of pulmonary fibrosis

Interestingly, a recent in vitro study in tissue slices showed that methionine pre-treatment displayed a protective effect against the toxic effects induced by MeHg and/or MeHg-Cys on mitochondrial function and cell viability, suggesting that methionine can be considered a potential strategy to the treatment of acute MeHg exposure (Roos et al., 2011)

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Methods: We synthesized highly effective FeSe 2 nanospheres using a one-step solvothermal process

Both of these effects mean you eat less food