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Axonal change in minor head injury

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Possible explanations are the reduced viability of chondrocytes, as found by Chu et al

Notably, rotenone is a mitochondrial complex I inhibitor that induces parkinsonism in animal models [92]

Its primary roles include: Neutralizing free radicals by increasing antioxidant enzymes like glutathione peroxidase, superoxide dismutase (SOD), and catalase Enhancing detoxification by stimulating Phase II enzymes such as glutathione S-transferase and NQO1, which help clear mycotoxins Protecting cellular DNA and mitochondria from oxidative and inflammatory damage Reducing inflammation by modulating the activity of inflammatory pathways like NF-B This response is critically important in conditions characterized by oxidative stresssuch as mold toxicity