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Studies have shown that supplementation can enhance fat use during exercise and increase stamina in athletic and unathletic people
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It is widely known that mitochondrial dysfunction (high ROS production, apoptotic cells, decreased m, and low cellular ATP levels) boosts the pathogenesis of KOA because it involves lipid homeostasis and cytokine release and promotes cell apoptosis [14, 15]
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Suormala T et al (2002) Biotin-dependent carboxylase activities in different CNS and skin-derived cells, and their sensitivity to biotin-depletion
