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Acknowledgements This work was supported by the Swedish Brain Foundation (grant FO2023-0241), the Swedish Research Council (VR-NT grant 2022-04198, VR-TVAR 2021-03293), and the Science for Life Laboratory (all to P.E
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doi: 10.1074/jbc.271.8.4209
Proc Natl Acad Sci U S A 105(48):1896418969
As the disease progresses, the body may experience impaired mitochondrial function, increased production of thiol proteins and free radicals, leading to disruptions in energy metabolism
