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DOI: 10.1038/s41598-026-43935-5

2022;50:W78290

*Correspondence: Santiago Navarro-Ledesma, [email protected] This article was submitted to Mitochondrial Research, a section of the journal Frontiers in Physiology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

Adduct formation of AcH with Cys or cysteinylglycine rather than GSH has also been suggested as a mechanism of EtOH-induced GSH depletion [50], however, the steady state levels of these thiols are much lower that for GSH in the liver

extreme thirst
