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Although the pathogenesis is not completely clear, the role of mucosal epithelial cell apoptosis, oxidative damage, retention of decomposed products, changes to the mast cells and other factors has aroused attention

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Similarly, a clinical study involving patients with coronary artery disease demonstrated that 12 weeks of L-carnitine supplementation resulted in reduced malondialdehyde levels, increased circulating L-carnitine concentrations, and enhanced activities of catalase and glutathione peroxidase enzymes compared to the placebo group