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Subsequently, OS causes further damage at cellular and subcellular levels, within the tissue microenvironment, and in endometrial function, thereby promoting the onset and progression of endometrial diseases

10.1016/j.carbpol.2014.12.037 145 ZhuJ.SunH.KangX.ZhuH.YanX

To test the hypothesis that a disorder of ROS homeostasis is induced by OsG-produced oxidative stress, an antioxidant, GSH (50 mg/kg/ 6 h), was subcutaneously injected into animals with LOsG challenge as described above, defined as the LOsG.TdGSH group ( n = 10)

PMID: 33 BindelsL

The temperature treatments constituted the second PCA (25.1%), which exhibited a distinct separation of metabolites affected by heat stress from those of the non-stress controls (Fig