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doi: 10.1016/j.jep.2016.01.001

He is also an engaged public historian, working closely with two UW campus initiatives researching the universitys histories of exclusion and resistance and its Native past and present

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At low concentrations or short durations of exposure, ozone, as an oxidative preconditioning, effectively enhances the antioxidant capacity with the help of H 2 O 2 , aldehydes, and other products.18 However, with a high concentration of exposure, the amount of ozone exceeds the buffering capacity of the blood, which directly damages cells and results in the production of many inflammatory factors and oxidative damage in a short period through its products.85,86 The acute damage can be respiratory, cardiovascular, or even fatal.124 The accumulation of H 2 O 2 and aldehydes produced by ozone at small dosages over extended periods of exposure can also cause injury

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Midbrain cultures were derived from E15 mice and had 3 H-DA uptake that was sensitive to GBR12909 at doses that were specific to DAT, indicating DAT-mediated uptake