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Alteration in innate immune response by Tlr9 and the Nalp3 inflammasome in acetaminophen-induced hepatotoxicity and induction of autophagy through the removal of damaged mitochondria and oxidative stress may be the potential mechanisms for aspirin-induced cytoprotection [15], [16]

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ACE2 was found more expressed in astrocytes (positive for glial fibrillary acidic protein, GFAP, green), and at a lower level in neurons (stained with -III-tubulin, red), as revealed by immunocytochemistry and confocal imaging analysis of 2-, 3- and 6-week old neurospheres (Fig