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Apr 28 2020;35(4):950-957

To this end, we inactivated genes encoding known or putative LD-associated proteins by RNAi

& Schofield, C

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In the liver, the accumulation of lipids is thought to impair mitochondrial function, thus reducing fatty acid oxidation and increasing the production of reactive oxygen species (ROS) that trigger lipid peroxidation, DNA damage, and apoptosis

Loss of function mutations in PCSK9 and drugs that inhibit PCSK9 result in increased LDL receptor activity and decreased LDL levels while gain of function mutations in PCSK9 lead to decreased LDL receptor activity and elevations in LDL levels